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1 Department of Medicine, Georgetown University School of Medicine, and Veterans Administration Hospital, Washington, D. C.
The mechanism of the diastolic pressure elevation occurring during left stellate ganglion stimulation was investigated. The cardiac output rose considerably, the heart rate remained essentially unchanged, and the total peripheral resistance fell moderately. The diastolic rise appeared to be due to increased blood flow rather than to any active changes in resistance vessels.
Submitted on June 22, 1962
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